Human endogenous retrovirus type K promotes proliferation and confers sensitivity to anti-retroviral drugs in Merlin-negative schwannoma and meningioma.

Authors

Sylwia Ammoun, Peninsula Medical School
Emmanuel A. Maze
Bora Agit
Shona Reeves
David A. Hilton, University Hospitals Plymouth NHS Trust
David B. Parkinson, Peninsula Medical School
Liyam Laraba, Peninsula Medical School
Emanuela Ercolano
Kathreena M. Kurian, University of Bristol
C. Oliver Hanemann, Peninsula Medical School
Robert D. Belshaw, Wenzhou-Kean University

ORCID

• Sylwia Ammoun: 0000-0002-1525-3583
• David B. Parkinson: 0000-0002-5704-4923
• C. Oliver Hanemann: 0000-0002-1951-1025

DOI Link

10.1158/0008-5472.can-20-3857

DOI

10.1158/0008-5472.can-20-3857

Publication Date

2021-12-01

Publication Title

Cancer Research

ISSN

0008-5472

Embargo Period

2022-12-01

Recommended Citation

Ammoun, S., Maze, E., Agit, B., Reeves, S., Hilton, D., Parkinson, D., Laraba, L., Ercolano, E., Kurian, K., Hanemann, C., & Belshaw, R. (2021) 'Human endogenous retrovirus type K promotes proliferation and confers sensitivity to anti-retroviral drugs in Merlin-negative schwannoma and meningioma.', Cancer Research, . Available at: 10.1158/0008-5472.can-20-3857